Vital Documents


CCSVI Information

Drugs, Side Effects, Safety, Dangers, and Financial Considerations

Since the middle of the 20th century with the advent of the ‘miraculous antibiotics’, most people have come to expect ‘drugs’ to solve their problems. Drugs often treat the symptoms but they may not treat the cause, Disease Modifying Drugs (DMDs) used in MS are a good example of this. By supressing the immune systems they may reduce the inflammation which produce some of the more unpleasant symptoms and may minimize some of the damage done by the scavenging white blood cells. But if we think of this disease not as an auto-immune disease as it has traditionally been thought of but as the result of venous problems then clearly these drugs are not treating the cause. And if that is the case then surely one of the side effects of these drugs may actually be making the disease process worse, by suppressing the immune system and opening the individual to attack from other organisms. As it is, side effects of these drugs can be pretty unpleasant of themselves. One in particular (Tysabri) has resulted in 58 deaths so far and 5 times that many serious and permanent worsening of patients’ condition. ( )

Chronic Cerebro-Spinal Venous Insufficiency (CCSVI) is a recently identified condition affecting the veins that drain the Central Nervous System (CNS). A large majority of people suffering from a collection of neurological symptoms named by the descriptive term Multiple Sclerosis (MS) because of the lesions that affect the brain and the spinal cord have been shown to have this condition. In short venous insufficiency means that the blood stays in the brain longer than it should and possibly causes damage through hypoxia, iron deposition and intracranial hypertension.

What about auto-immunity?

Historically MS researchers reported that lesions were venocentric, it is only in the past 40 years that the theory of auto-immunity has come to the fore; largely due to the development of an animal model of MS - Experimental Autoimmune Encephalomyelitis (EAE).

However EAE is not MS and a number of studies referenced below describe the differences between the two which not only makes it difficult to draw conclusions from research using this model but also casts doubt on MS being an auto-immune disease. Undoubtedly the immune system is involved in the disease process but macrophage activity is largely an innate scavenging response to the presence of degenerate and dead myelin. (A P D Henderson et al, 2009)

A couple of extracts discussing the theories of MS. You can read the full text in this pdf: